top of page

Coffee – The good, the bad and the caffeinated

Coffee receives all sorts of conflicting press and for good reason. Like many substances, there are benefits and a down side to your morning beverage.

The effect of coffee will depend on factors that are attributed to the coffee itself and the individual consuming it. (refer to Figure 1). Short term effects are probably more well known than the long term ones and this is because the latter is harder to measure and predict. Additionally, the evidence is not quite to where it needs to be, but is pointing towards benefits from drinking small amounts.

It is likely that reading the evidence available on coffee vs health won’t change your mind about drinking it. It might however initiate thought about the quantity you drink as well when & why you drink it.


What we do know


Negatives of coffee consumption:

  • Can interrupt sleep patterns

  • Can exacerbate fatigue

  • May increase blood pressure

  • Not recommended during pregnancy and breastfeeding

  • Encourages a reliance on caffeine consumption

  • Withdrawal can be uncomfortable

  • Consumption is reported by people with many different serious health conditions

  • Poorly tolerated by some individuals with a specific genetic marker

  • More research is needed to identify any links to long term health issues

On the positive side:

  • Gets your bowels moving

  • Improves mood

  • Helps with alertness / counteracts sleepiness

  • No proof that it increases the risk of any type of cancer

  • Contains antioxidants which is promising for neurological disorders

  • It is not as acidic as expected – sitting around pH 5

  • Improves symptoms in Parkinson’s patients

  • Can improve sports performance short term

When looking at the copious amounts of research, it seems that drinking larger amounts (estimated to be anything over 4 cups a day), may lead to health concerns. When consumed in modest amounts, there is little to no effect on health, while others drinking small amounts may be of benefit long term. Additives to your coffee such as milk and sugar are also going to change things slightly. Milk adds some essential nutrients while sugar doesn't provide any health benefits.


How can coffee be used effectively?


1. Don’t use it to mask other problems.

If coffee is being used to overcome constant tiredness in the morning - review your sleep habits. Relying on coffee to function is like your car always having an almost empty gas tank. Only petrol can replenish the fuel supply, nothing else. Coffee can't replace sleep. Getting at least 8 hours sleep will refill your tank.


2. Get the timing right.

Caffeine will always affect the sleep/wake cycle no matter what time it is consumed. The closer to sleep, the more likely it will have an adverse affect. Many experts say to cut yourself off by 2pm, but often I advise people to stop at midday especially where there are extreme sleep and fatigue problems.

If you get to your cut-off time and haven’t had your coffee, consider skipping it or reducing your normal quantity of cups.


3. Adjust accordingly

Your regular coffee routine might not sit well every day so adjust as necessary. If you wake up refreshed, consider your need for it that day or consider consuming less. It’s often we drink it out of habit instead out of necessity.


For more targeted advice suited to you, including assistance with Nutrition and Dietary choices, please get in touch via my website, via phone or email at enquiries@purpletreetherapies.com.au


Purple Tree Therapies is located in New Farm but I can also do video consultations via Zoom.


References

Pourshahidi, L. K., Navarini, L., Petracco, M. & Strain, J.J. (2016). A Comprehensive Overview of the Risks and Benefits of Coffee Consumption. Institute of Food Technologists. 15. 671 – 684: doi: 10.1111/1541-4337.12206


Arab L, Biggs ML, O’Meara ES, Longstreth WT, Crane PK, Fitzpatrick AL. (2011). Gender differences in tea, coffee, and cognitive decline in the elderly: the cardiovascular health study. J Alzheimer’s Dis 27, 553–66


Awaad AS, Soliman GA, Al-Outhman MR, Al-Shdoukhi IF, Al-Nafisah RS, Al-Shamery J, Al-Samkhan R, Baqer M, Al-Jaber NA. (2011). The effect of four coffee types on normotensive rats and normal/hypertensive human volunteers. Phytotherapy Res 25:803–8.


Bakuradze T, Boehm N, Janzowski C, Lang R, Hofmann T, Stockis J-, Albert FW, Stiebitz H, Bytof G, Lantz I, Baum M, Eisenbrand G. 2011. Antioxidant-rich coffee reduces DNA damage, elevates glutathione status and contributes to weight control: results from an intervention study. Mol Nutr Food Res, 55:793–7.


Bakuradze T, Parra GAM, Riedel A, Somoza V, Lang R, Dieminger N, Hofmann T, Winkler S, Hassmann U, Marko D, Schipp D, Raedle J, Bytof G, Lantz I, Stiebitz H, Richling E. (2014). Four-week coffee consumption affects energy intake, satiety regulation, body fat, and protects DNA integrity. Food Res Int 63:420–7


Barrett-Connor E, Chang JC, Edelstein SL. 1994. Coffee-associated osteoporosis offset by daily milk consumption: the Rancho Bernardo study. J Am Med Assoc, 271:280–3


Boobis A, Chiodini A, Hoekstra J, Lagiou P, Przyrembel H, Schlatter J, Schutte K, Verhagen H, Watzl B. 2013. Critical appraisal of the assessment of benefits and risks for foods, ‘BRAFO Consensus Working Group’. Food Chem Toxicol, 55:659–75


Buscemi S, Verga S, Batsis JA, Donatelli M, Tranchina MR, Belmonte S, Mattina A, Re A, Cerasola G. 2010. Acute effects of coffee on endothelial function in healthy subjects. Eur J Clin Nutr, 64:483–9.


Camfield DA, Silber BY, Scholey AB, Nolidin K, Goh A, Stough C. (2013). A randomised placebo-controlled trial to differentiate the acute cognitive and mood effects of chlorogenic acid from decaffeinated coffee. PLoS One 8:e82897.


Cano-Marquina A, Tarin JJ, Cano A. (2013). The impact of coffee on health. Maturitas 75, 7–21.

Conde A, Teves C, Figueiredo B. (2011). Maternal coffee intake and associated risk factors: effects on fetal growth and activity. Acta Med Port, 24, 241–8.


Espinosa J, Rocha A, Nunes F, et al. Caffeine consumption prevents memory impairment, neuronal damage, and adenosine A2A receptors upregulation in the hippocampus of a rat model of sporadic dementia. (2013). J Alzheimers Dis. 34, 509‐518.


European Food Safety Authority (EFSA). 2015. Scientific opinion on the safety of caffeine. EFSA J 13:4102. doi:10.2903/j.efsa.2015.4102


Fardet A, Boirie Y. 2014. Associations between food and beverage groups and major diet-related chronic diseases: an exhaustive review of pooled/meta-analyses and systematic reviews. Nutr Rev, 72, 741–62.


Green CJ, de Dauwe P, Boyle T, Tabatabaei SM, Fritschi L, Heyworth JS. (2014). Tea, coffee, and milk consumption and colorectal cancer risk. J Epidemiol 24, 146–53.


Popat RA, Van Den Eeden, SK, Tanner CM, Kamel F, Umbach DM, Marder K, Mayeux R, Ritz B, Ross GW, Petrovitch H, Topol B, McGuire V, Costello S, Manthripragada AD, Southwick A, Myers RM, Nelson LM. (2011). Coffee, ADORA2A, and CYP1A2: the caffeine connection in Parkinson’s disease. Eur J Neurol, 18, 756–65


Rivera‐Oliver M, Diaz‐Rios M. Using caffeine and other adenosine receptor antagonists and agonists as therapeutic tools against neurodegenerative diseases: a review. (2014). Life Sci. 101, 1‐9


Nabbi‐Schroeter D, Elmenhorst D, Oskamp A, Laskowski S, Bauer A, Kroll T. Effects of long‐term caffeine consumption on the adenosine A1 receptor in the rat brain: an in vivo PET study with [(18)F] CPFPX. (2018). Mol Imaging Biol, 20, 284‐291.


Urry E, Landolt HP. Adenosine, caffeine, and performance: from cognitive neuroscience of sleep to sleep pharmacogenetics. (2015). Top Behav Neurosci. 25, 331‐366

Single Post: Blog_Single_Post_Widget
bottom of page